Aryl hydrocarbon receptor ligand 2,3,7,8-tetrachlorodibenzo-p-dioxin
enhances liver damage in bile duct-ligated mice.
Gestational exposure to the AhR agonist 2,3,7,8-tetrachlorodibenzo-p-dioxin
induces BRCA-1 promoter hypermethylation and reduces BRCA-1 expression in mammary tissue of rat offspring: preventive effects of resveratrol.
alters the differentiation pattern of human keratinocytes in organotypic culture.
Seveso Women's Health Study: a study of the effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin
on reproductive health.
Biochemical and toxic effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin
in immature male and female chickens.
KEY WORDS: acetylcholinesterase (AChE), aryl hydrocarbon receptor (AhR), dioxin-responsive element (DRE), neuron, 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD), transcriptional regulation.
Microarray analysis of early adipogenesis in C3H10T1/2 cells: cooperative inhibitory effects of growth factors and 2,3,7,8-tetrachlorodibenzo-p-dioxin
The estimation of elimination rates of persistent compounds: a reanalysis of 2,3,7,8-tetrachlorodibenzo-p-dioxin
levels in Vietnam veterans.
Glucocorticoid potentiation of cytochrome P4501A1 induction by 2,3,7,8-tetrachlorodibenzo-p-dioxin
in porcine and human endothelial cells in culture.
In utero 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD) alters reproductive morphology and function in female rat offspring.
Molecular and cellular mechanisms of 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD), the most potent compound of PCDD/Fs, are well characterized.
Investigations on the effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin
(TCDD) on parameters of various immune functions.