ACBP

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AcronymDefinition
ACBPAtlantic City Beach Patrol (New Jersey)
ACBPAorto-Coronary Bypass (cardiac surgery)
ACBPAdvanced Concepts Base Program
ACBPAcyl-CoA (Coenzyme A) Binding Protein (molecular biology)
ACBPAutologous Cord Blood Plasma
References in periodicals archive ?
a) The upper panel is a Western Blot showing expression levels of Acyl-CoA Synthetase (ACSL1) and Acetyl-CoA Synthetase (ACeCS1) with respective contents of [beta]-actin.
16) When viewed with the addition of fish oil, both the peroxisomal palmitoyl-CoA oxidation rate and acyl-CoA oxidase activity levels were distinctly higher.
La liberacion de tritio unido a los carbonos 9 y 10 del palmitato depende de tres mecanismos: 1) la accion de una acyl-CoA deshidrogenasa formando un 2,3-enoyl-CoA ester y transfiriendo el 50% del tritio a la proteina de transferencia de electrones (flavina adenina dinucleotido reducida, FADH); 2) la reaccion de la 3-hidroxiacil-CoA deshidrogenasa que remueve la mitad del tritio remanente, el cual finaliza en la nicotinamida adenina dinucleotido (NADH); 3) el ciclo del acido tricarboxilico que libera el 25% restante.
Medium-chain acyl-CoA dehydrogenase (MCAD) deficiency: diagnosis by acylcarnitine analysis in blood.
For example, one of the disorders included in the expanded panel, medium-chain acyl-CoA dehydrogenase deficiency (MCAD), involves a simple treatment (i.
People who fast for one or two days experience an increase in fatty acyl-CoA levels.
and his laboratory at Brigham and Women's Hospital, demonstrated that Them1, a long chain fatty acyl-CoA thioesterase that is highly enriched in brown fat, plays an important role in regulating energy homeostasis.
Additionally, acyl-CoA dehydrogenase, very long chain, which is involved in a fatty acid [beta]-oxidation pathway, was found in the QTL interval on SSC12 [14].
avoid fasting in medium-chain acyl-CoA dehydrogenase deficiency (MCAD) and long-chain l-3-hydroxyacylCoA dehydrogenase deficiency (LCHAD)], and vaccination (e.
Background: Late-onset multiple acyl-CoA dehydrogenase deficiency (MADD) is the most common type of lipid storage myopathies in China.
Fatty acid anion results from the hydrolysis of accumulated fatty acyl CoA by mitochondrial acyl-CoA thioesterase-1 (MTE-1) to liberate free CoA required for continued fatty acid oxidation.
Hepatic fatty acid synthase (FAS) and Acyl-CoA oxidase (AOX) activities were lower and higher in IUGR piglets respectively.
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