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C5a |
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At the time of over-activation by malaria infection, C5a appears leads to an excessive inflammatory response and disrupts normal blood vessel growth in the placenta, raising the risk of spontaneous abortions or low birth weight infants. Essentially most extracellular virulence factors, including M protein, streptokinase, SpeF, Sda1, C5a peptidase, and the secreted inhibitor of complement, are degraded by this protease, resulting in decreased virulence. Komplemanin aktivasyonu sonucu proinflamatuvar urunlerinden C3, C5a ve C5b aciga cikar. |
C5a |
C4ORF1 C4P C4PH C4plant C4PM C4PR C4QL C4RD C4RDP C4RG C4S C4SC C4SMP C4SSR C4T C4U C4USM C4VM C4W C4Y C5 C5 C5-A C50 C57Bl/6 Mouse Embryo Fibroblast C5a C5a receptorC5aR C5CC C5F C5H C5I C5I2 C5IMP C5ISR C5O C5R C5RA C5SC C6 C6 C6 oxidative pathway C60 C60 C60 C64 C64 C65 c68 C6F C6G C6h C6H12O6 C6h5coona C6h5coona C6h5coona | |||||
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