HaCaTCultured Human Keratinocyte (cells)
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Six most up-regulated proteins in HaCaT against DNBS stimulation were chromosome X ORF 26 (Cxorf26), co-chaperone P23 (PTGES3), calmodulin (CALM3), interferon-gamma inducing factor precursor (IL-18), smooth muscle/non-muscle myosin alkali light chain (MYL6)and breakpoint cluster region protein 1 (BANF1).
Aberrant heterodimerization of keratin 16 with keratin 6A in HaCaT keratinocytes results in diminished cellular migration.
The cell lines used were HaCaT cells (Three genetically altered HaCaT derived cell lines were used provided most generously by Prof.
Silibinin is a potent sensitizer of UVA radiation-induced oxidative stress and apoptosis in human keratinocyte HaCaT cells.
The plant has been shown to protect against UVB-induced HaCaT keratinocyte damage [3].
The extract was also seen to absorb UVA and UVB irradiation and demonstrated a dose-dependent protection of HaCaT human keratinocytes and mouse fibroblasts 3T3 cells against UVB-induced cytotoxicity.
Human papillomavirus-16 E7 interacts with Siva-1 and modulates apoptosis in HaCaT human immortalized keratinocytes.
Galactosyl ceramide localized primarily to the plasma membrane of HaCaT H-3 keratinocytes but was predominantly intracellular in other epithelial cells.
Cytotoxicity of resin monomers on human gingival fibroblasts and HaCaT keratinocytes.
Peroxidated squalene induces the production of inflammatory mediators in HaCaT keratinocytes: a possible role in acne vulgaris.
Pharmacological inhibition of TRPC1 channels reduced the migrating potential of HaCaT cells in vitro in response to growth factor stimulation.