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The role of the KOR in butorphanol-induced PVN neuronal activation was explored by intracerebroventricular administration of nor-binaltorphimine (nor-BNI), prior to butorphanol (10 mg/kg) administration. The observation of Turton et al (74) that intracerebroventricular (IVC) application of GLP-1 was a powerful inhibitor of food intake in rats, whereas intraperitoneal (IP) administration of the peptide did not have any effect suggested a centrally mediated pathway. Male Sprague-Dawley rats received intracerebroventricular (i. |
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