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TRAF2TNF Receptor-Associated Factor 2
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They tested this discovery in 55 human samples of pancreatic cancer, and found that in 69 percent, TRAF2 wasn't functioning properly and there were higher levels of other molecules participating in the alternative pathway
10-14,23] Finally, the overexpression of E2F-1 blocks antiapoptotic signal pathways such as NF[Kappa]-B and JNK/SAPK activation,[48] by down-regulating TRAF2 gene expression, a regulator of NFK-B and JNK/SAPK activity.