The thiobarbituric acid-reactive substance
(TBARS) content was determined as reported by Lemon (1975) and Kilic and Richards (2003).
Lipid peroxidation measured as thiobarbituric acid-reactive substance
in tissue slices: Characterization and comparison between homogenates and microsome.
At the conclusion of the storage period, the investigators determined the amount of thiobarbituric acid-reactive substance
in the samples as an indicator of lipid oxidation.
The absorbance of thiobarbituric acid-reactive substance
was determined at 535 nm.
During storage, the researchers performed thiobarbituric acid-reactive substance
(TBARS) and microbial analysis of the samples.
Furthermore, the serum thiobarbituric acid-reactive substance
level decreased after oral administration of Coptidis Rhizoma extract, indicating that Coptidis Rhizoma could prevent hypercholesterolemic disease through reducing lipid peroxidation.
The oxidative stability of the mozzarella lipid fraction and governing liquid were measured by the dosage of thiobarbituric acid-reactive substances
(TBARs)  and expressed in [micro]g of malonyldialdheyde (MDA) per g of fat and [micro]g of MDA per mL, respectively.
The researchers evaluated the berries' physicochemical characteristics for Brix, percentage of moisture content, pH value, color (L*, a* and b* values) and lipid oxidation potential, using the thiobarbituric acid-reactive substances
(TBARS) protocol every seven days for 28 days.
Intestinal thiobarbituric acid-reactive substances
and myeloperoxidase levels and tissue damage scores for the intestines in the thioacetamide+methylprednisolone group were lower than those in the thioacetamide group (p<0.
Attack of the hydroxyl radicals on deoxyribose led to formation of thiobarbituric acid-reactive substances
(TBARS) which were measured by the method of Ohkawa et al.
Lipid peroxidation and reactive oxygen species (ROS) formation were determined by thiobarbituric acid-reactive substances
(TBARS) and 2'-7'-dichlorofluorescein (DCF), respectively.
Administration of NMDA, causing a lethality of approximately 60%, resulted in a significant decrease of total glutathione (GSH) level and increase of thiobarbituric acid-reactive substances
(TBARS) value in brain tissue.