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Related to URIC: gout
URICUniversity Research Instrumentation Center (University of Arizona, Tucson, AZ)
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In contrast to numerous studies demonstrating uric acid mobilization from fat body of cockroaches (Cochran 1975), only limited study has been performed in this area with termites (Breznak 2000).
We wanted to investigate if serum uric acid levels are independently associated with CACS in patients without known CHD, and thus provide support for the reliability of serum uric acid level as an additional test in cardiovascular risk assessment.
Dr Kumar said excess uric acid in the body is converted into crystals which then get deposited in the joints.
You can also help prevent gout attacks by losing weight but avoid diets that are high in protein as these can raise uric acid levels.
Uric acid levels from feces and 3 groups of 10 whole termites were determined using a procedure detailed in Arquette & Forschler (2006).
In order to measure the uric acid in the urine sample, first samples were diluted to become readable for the spectrophotometer.
It is possible to decrease the uric acid pool or prevent it from accumulating.
The patients meeting the following criteria were excluded from the study: renal artery stenosis, coarctation of the aorta, pheochromocytoma, primary aldosteronism, Cushings's syndrome, renal disease, and increased dietary-sodium intake/drugs, or those that were taking drugs that interfere with uric acid levels and/or those with a history of gout or renal stones.
We are now able to measure ascorbic acid, dopamine and uric acid in biological and pharmaceutical liquids simultaneously thanks to the modification made to carbon-ceramic electrodes by carbon nanotubes," Biyouk Habibi, member of scientific board of chemistry department at Tarbiat Moallem University of Azarbaijan, told INIC.
The researchers found that blood levels of uric acid and C-reactive protein (a marker for inflammation) significantly decreased in patients treated with allopurinol.
Such a conclusion is supported mechanistically by in vitro studies by Mazzali and coworkers (with RJ Johnson) (4) and other investigators, (5,6) who have demonstrated that soluble uric acid is a biologically active molecule that has potentially pro-hypertensive effects on both vascular endothelium and the kidney.
The source of the pain associated with gout is a build-up in the blood of uric acid, which can form sharp crystals that collect in the joints.