Acronym	Definition
APCR	Activated Protein C Resistance
APCR	Air Pollution Control Residue
APCR	Armor Piercing Composite Rigid
APCR	Association pour la Catéchèse en Rural (French: Association for Rural Catechism)
APCR	Analysis, Prompting, Checking, and Reporting
APCR	Activate PDP (Packet Data Protocol) Context Request
APCR	Association Pêche Côtière du Roussillon (French: Coastal Fisheries Association of Roussillon; Roussillon, France)
APCR	Anchored Polymerase Chain Reaction (genetics)
APCR	Association de la Presse Catholique Régionale (French: Regional Catholic Press Association)
APCR	Asymmetric Polymerase Chain Reaction (hematology)

References in periodicals archive

The clinical application of a new specific functional assay to detect the factor V(Leiden) mutation associated with activated protein C resistance. Am J Clin Pathol.

Modified test for activated protein C resistance. Lancet.

Rivaroxaban Causes Missed Diagnosis of Protein S Deficiency but Not of Activated Protein C Resistance (Factor V Leiden)

The discovery of activated protein C resistance. J Thromb Haemost 2003 Jan; 1(1):3-9.

A 52-year-old man with ecchymotic leg ulcers

Today, IL offers a comprehensive line of HemosIL reagents for the diagnosis of thrombophilia, including antithrombin, protein C, protein S, activated protein C resistance, factor V Leiden, homocysteine, ThromboPath, and Xpert HemosIL FII & FV molecular.

Instrumentation Laboratory wins patent infringement and unfair competition trial against Diagnostica Stago relating to reagents for hemostasis

Activated protein C resistance (FV Leiden) and thrombosis: factor V mutations causing hypercoagulable states.

Factor V Leiden: an overview

Activated protein C resistance phenotype and genotype in patients with primary antiphospholipid syndrome.

Activated protein C resistance phenotype in patients with antiphospholipid antibodies.

Acquired activated protein C resistance associated with IgG antibodies against [[beta].sub.2]-glycoprotein I and prothrombin as a strong risk factor for venous thromboembolism

Activated protein C resistance is typically assessed by mixing patient plasma with factor V-deficient plasma.

Evaluating idiopathic venous thromboembolism: what is necessary, what is not

Table 1 Genetic causes of venous thrombosis Common Activated protein C resistance (APCR)/ Factor V Leiden Prothrombin 20210 mutation Methylenetetrahydrofolate reductase gene mutation Increased factor VIII activity Rare Deficiencies of antithrombin and proteins C and S Very Rare Dysfibrinogenemia Table 2 Acquired conditions affecting functional hypercoagulability test results Antithrombin Decreased in heparin use, recent thrombosis, liver disease, DIC, (1) nephrotic syndrome APCR First generation assays: High factor VIII level, LA, (2) acute phase response Second generation assays: Strong LA, (2) pregnancy Factor VIII Elevated in acute phase reaction; falsely decreased in the presence of LA (2) Homocysteine Elevated in recent thrombosis.

Testing for genetic predisposition to venous thrombosis

The Clinical Significance of Activated Protein C Resistance

Activated protein C resistance, the factor V Leiden mutation, and a laboratory testing algorithm. (Original Articles)

Evaluation of a tissue factor dependent factor V assay to detect factor V Leiden: demonstration of high sensitivity and specificity for a generally applicable assay for activated protein C resistance. Br J Haematol 1996;95:550-3.

Multicenter evaluation of PCR methods for the detection of factor V Leiden (R506Q) genotypes

Evaluation of a tissue factor V assay to detect factor V Leiden: demonstration of high sensitivity and specificity for a generally applicable assay for activated protein C resistance. Br J Haematol 1996;95:550-3.

Factor V Leiden and other coagulation factor mutations affecting thrombotic risk