HFE

(redirected from hemochromatosis)
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AcronymDefinition
HFEHemochromatosis
HFEHuman Factors Engineering
HFEHydrofluoroether (cleaning solvent)
HFEHeavy Fuel Engine (unmanned aerial vehicles)
HFEHuman Factors in Electronics
HFEHope For Europe
HFEHorizontal Fiscal Equalisation
HFEHeat Flow Experiment
HFEForward Current Gain
HFEHuman Failure Event
HFEHelmholtz Free Energy
HFEHardware Forwarding Engine (Cisco)
HFEHigh Fuel Economy
HFEHandicap FormEduc (Louga, Senegal)
HFEHeavy Front Engine
HFEHigh Frequency Engineering
HFEHerschend Family Entertainment Corporation (theme parks; Norcross, GA)
References in periodicals archive ?
The report provides a snapshot of the global therapeutic landscape of Hemochromatosis
Patients with hereditary hemochromatosis may be asymptomatic or may present with generalized weakness.
Liver disease: Hemochromatosis liver disease resembles and exacerbates viral hepatitis, alcoholic hepatitis, and porphyria.
Clinical consult: iron overload-hereditary hemochromatosis.
The term hemochromatosis encompasses all diseases with iron accumulation disorders.
But only those who inherit two C282Y mutations, one from their mother and one from their father, are at risk for hemochromatosis, which is full-blown iron overload.
Mutations of the HFE gene are found in nearly 90% of patients with clinical hemochromatosis, most of whom are homozygous (2).
Genetic testing revealed that the patient was homozygous for the C282Y mutation of the HFE gene, confirming a postmortem diagnosis of hereditary hemochromatosis (4).
In contradiction, hemochromatosis preferentially deposits iron in the pituitary gland.
Medical College of Georgia researchers are pursuing a link between hemochromatosis, which results in iron overload, and the wet form of macular degeneration, the leading cause of blindness in people 60 and older.
Hemochromatosis is a serious condition that is a result of untreated iron overload.
The have organized the papers into sections that focus in turn on: chemistry, biochemistry, and the formation and reactivity of endogenous toxins such as metabolic intermediates and reactive oxygen species, particularly those associated with excessive sugar, fat, meat, or alcohol consumption; the association of increased endogenous toxin levels with inborn errors of metabolism such as galactosemia, hyperlipidemia, porphyria, hemochromatosis, and related conditions; examples of endogenous toxins that appear to be associated with acquired diseases or animal disease models, including hepatic injury, asthma, rheumatism, colorectal cancer, reperfusion diseases, neurodegeneration, and aging; and nutritional and pharmacologic therapeutics that have been proposed for decreasing endogenous toxins.