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PBGPepsi Bottling Group (stock symbol)
PBGPoint-Based Graphics
PBGPhotonic Bandgap
PBGProgram Budget Guidance
PBGPostprandial Blood Glucose
PBGProgram and Budget Guidance
PBGPrime Bank Guarantee
PBGPerformance Bond Guarantee
PBGPersonal Branch Gateway (Pannaway)
PBGProduct Business Group
PBGPlanned Building Group
PBGPrzedsiebiorstwo Badan Geofizycznych (Geophysical Exploration Company, Poland)
PBGPie Bill Gates (game)
PBGPretty Bloody Good
PBGPilot Boarding Ground (naval travel; various nations)
PBGParallel Blade Ground (electrical connection)
PBGPressure Breathing for Gravity
References in periodicals archive ?
Acute intermittent porphyria (AIP) is a rare metabolic disorder that is the most common of the acute porphyries and is characterized by enzymatic defect of porphobilinogen deaminase with depot and increased excretion of porphyrins and their precursors (1).
Allosteric inhibition of human lymphoblast and purified porphobilinogen deaminase by protoporphyrinogen and coproporphyrinogen.
Chlorophyll biosynthesis in higher plants was carried and accomplished by sequential reactions, D-aminolevulinic acid (ALA), porphobilinogen (PBG), uroporphyrinogen III (Urogen III), coproporphyrinogen III (Coprogen III), protoporphyrin IX (Proto IX), Mg-protoporphyrin IX (Mg- Proto IX) and protochlorophyllide (Pchlide) were the major synthetic precursors during these sequential reactions (Ilag et al.
4] During an acute attack, haem precursors accumulate in front of the deficient enzyme, which in AIP is porphobilinogen deaminase and in VP protoporphyrinogen oxidase.
1 Acute intermittent porphyria (AIP), an autosomal dominant disorder, is a common type of neurologic porphyria in which mutation of the porphobilinogen deaminase (PBGD) gene plays an important role.
Mutation hotspots in the human porphobilinogen deaminase gene: Recurrent mutations G111R and R173Q occurring at CpG motifs.
For confirmatory diagnosis, porphobilinogen deaminase (PBGD) test was done which was found to be positive.
Thus, N deficiency leads to decreased synthesis of glutamate, reduced ALA porphobilinogen synthesis and, consequently, a decrease in the biosynthesis of chlorophyll, which leads to the development of chlorosis in plants (CHU et al.
3-5) Test for urinary porphobilinogen was done by the method of Schwartz et al4 and was negative.
Urine examination was normal including porphobilinogen.