AHLH

AcronymDefinition
AHLHAckerman Handcrafted Log Homes (Carbondale, CO)
AHLHAccredited Home Lenders Holding Co. (Washington, DC)
References in periodicals archive ?
The study included 44 unrelated AHLH patients treated at 2 hospitals (42 from Hacettepe University and 2 from Kirikkale University) between 2005 and 2009, and 164 healthy controls.
The etiologic agents in the AHLH patients are shown in Table 1.
Etiologic agents and perforin A91V transition in the AHLH patients Underlying Disease Number of A91V mutation Patients positive EBV infection 6 2 Leukemia 5 Autoimmune disease 4 Combined E.
In all, 5 (11.3%) of the AHLH patients were heterozygous for the A91V transition (Table 2).
Distribution of perforin gene A91V transition, and TNF-cx promoter-1031 T>C polymorphism genotypes and alleles Group Perforin TNF - A91V [alpha] - 1031 T>C Genotype Allele Total A91V+ Total CC TC TT C Studied n (%) Studied n (%) n (%) n (%) n (%) (n) (n) AHLH 44 5 40 1 16 23 18 (11.3) (2.5) (40) (57.5) (22.5) Control 159 7 164 7 58 99 72 (4.4) (4.3) (35.4) (60.3) (22) Group T n (%) AHLH 62 (77.5) Control 256 (78) TNF[alpha] promoter-1031 T > C polymorphism
Among the 40 AHLH patients examined for the polymorphism, 1 (4%) had the CC genotype and 16 (40%) had the TC genotype.
In the present study 5 (11.3%) AHLH patients with infection had A91V in the heterozygote state, suggesting that the transition may be a predisposing factor for infection-associated AHLH; the difference between the 2 groups was not statistically significant.