Upon electro diagnostic studies and examination, the GBS variants distribution was as follows: Acute inflammatory demyelinating polyradiculo-neuropathy variant (AIDP
) in 49 (39.2%) of the patients, acute motor axonal neuropathy (AMAN) variant in 67 (53.6%), 6(4.8%) patients having mixed (axonal + demyelinating) variety as shown in table-IIII.
The most frequent type was AIDP
followed by, with descending order, acute motor sensory axonal, acute motor axonal, MFS, pure sensory type;
In our study, patients with involvement of sensory as well as motor systems on the electrodiagnostic tests i.e., patients with the AMSAN variant showed delayed recovery and residual disability compared to the AMAN and AIDP
variants where there was spontaneous recovery.
Mean CD4 count (in cell/uL) in this study was 151+-100.9 with DSPN, 259.7+-113.9 with AIDP
, 120.3+-81.8 with TBM, 42.0+-29.60 with cryptococcal meningitis, and 54.6+-38.7 with toxoplasmosis.
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It includes AIDP
, AMAN, acute motor-sensory axonal neuropathy (AMSAN), Miller Fisher syndrome, acute panautonomic neuropathy and acute sensory neuropathy, and other subtypes.
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Acute inflammatory demyelinating polyneuropathy (AIDP
) and acute motor axonal neuropathy (AMAN) are the common subtypes of GBS [1, 2].
Complications of GBS vary among subtypes, which can be mainly acute inflammatory demyelinating polyneuropathy (AIDP
), Acute motor axonal neuropathy (AMAN), Acute motor sensory axonal neuropathy (AMSAN), and Miller-Fisher syndrome (MF) [2, 3].
Acute Inflammatory Demyelinating Polyneuropathy (AIDP
) is the most common variant of Guillain-Barre syndrome in the United States.
These findings suggested a probable AIDP
triggered by WNV.