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References in periodicals archive ?
Protective effect of resveratrol against acute lung injury induced by lipopolysaccharide via inhibiting the myd88-dependent Toll-like receptor 4 signaling pathway.
Acute lung injury and the acute respiratory distress syndrome: a clinical review.
Transfusion- related acute lung injury. Ind J Pathol Microbio 2009;52:561-3.
Lipoxin A4 attenuates LPS-induced mouse acute lung injury via Nrf2-mediated E-cadherin expression in airway epithelial cells.
Zhang, "Vitamin D alleviates lipopolysaccharideinduced acute lung injury via regulation of the reninangiotensin system," Molecular Medicine Reports, vol.
Obara, "Lidocaine attenuates acute lung injury induced by a combination of phospholipase A2 and trypsin," Critical Care Medicine, vol.
The MCP-1/CCR2 pathway is only one of many pathways in systematic disease such as acute lung injury. LPS is recognized by toll-like receptors on the membrane of antigen-presenting cells, which triggers LPS/TLR/MyD88/IRAK/TRAF/NF-[kappa]B signaling pathway [33, 34].
Liu, "Hypoxia-induced acute lung injury is aggravated in streptozotocin diabetic mice," Experimental Lung Research, vol.
Bertolini et al., "Epidemiology and outcome of acute lung injury in European intensive care units.
For example, in a murine blunt chest injury model with lung contusion, neutrophils and their oxidative response have been identified as a major contributor to acute lung injury and neutrophil depletion was protective [155].
[2-4] Since the 1970s, death of patients with paraquat poisoning has been identified as mainly associated with acute lung injury. [5]
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