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References in periodicals archive ?
Oak et al., "Absence of TNF-[alpha] enhances inflammatory response in the newborn lung undergoing mechanical ventilation," American Journal of Physiology Lung Cellular and Molecular Physiology, vol.
Heller, "Persistence of circadian rhythmicity in hibernating ground squirrels," The American Journal of Physiology, vol.
Badoer, "Hypothalamic paraventricular nucleus is critical for renal vasoconstriction elicited by elevations in body temperature," American Journal of Physiology Renal Physiology, vol.
Bakajsova, "Protein kinase C-a activation promotes recovery of mitochondrial function and cell survival following oxidant injury in renal cells," American Journal of Physiology Renal Physiology, vol.
Crisostomo et al., "Endothelial STAT3 plays a critical role in generalized myocardial proinflammatory and proapoptotic signaling," American Journal of Physiology Heart and Circulatory Physiology, vol.
Hood, "Calcium-regulated changes in mitochondrial phenotype in skeletal muscle cells," American Journal of Physiology. Cell Physiology, vol.
Mustafa, "High-salt diet enhances mouse aortic relaxation through adenosine [A.sub.2A] receptor via CYP-epoxygenases," American Journal of Physiology - Regulatory Integrative and Comparative Physiology, vol.
The study, "Prolonged Sitting-induced Leg Endothelial Dysfunction is Prevented by Fidgeting," recently was published by the American Journal of Physiology Heart and Circulatory Physiology.
American Journal of Physiology Regulatory Integrative and Comparative Physiology 301, R67-R75.
The researchers, writing in the American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, said: "Exercise can be performed at a given workload for a longer period of time before the onset of fatigue."
Citation: Christa Nicole Grant et al.; "Human and Mouse Tissue-Engineered Small Intestine Both Demonstrate Digestive And Absorptive Function"; American Journal of Physiology: GI & Liver, January 2015 DOI: 10.1152/ ajpgi.00111.2014
Rodriguez-Iturbe et al., "Angiotensin II, interstitial inflammation, and the pathogenesis of salt-sensitive hypertension," American Journal of Physiology: Renal Physiology, vol.
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