(redirected from Atypical Teratoid/Rhabdoid Tumor)
ATRTAtypical Teratoid/Rhabdoid Tumor
ATRTAll The Right Type
ATRTAll Terrain Recon Transport (Star Wars)
ATRTAutomated Test and Re-Test (software testing; US Navy)
ATRTAutomation and Technology Round Table (Texas Library Association)
ATRTAnti-Transmit/Receive Tube
ATRTAverage Token Rotation Time
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References in periodicals archive ?
Imaging findings of CNS atypical teratoid/rhabdoid tumors. AJNR Am J Neuroradiol 2004;25(3):476-480.
Primary intracranial atypical teratoid/rhabdoid tumors of infancy and childhood: MRI features and patient outcomes.
INI1 protein expression distinguishes atypical teratoid/rhabdoid tumor from choroid plexus carcinoma.
Nonsense mutation and inactivation of SMARCA4 (BRG1) in an atypical teratoid/rhabdoid tumor showing retained SMARCB1 (INI1) expression.
Comparison of Diagnostic Agreement Among Reference Pathologists Reviewing Hematoxylin/Eosin Slides of Representative Blocks of Tumor From the Entire Cohort of 96 Patients Versus the Referring Institutions' Diagnoses as Either Medulloblastoma (MB) or Atypical Teratoid/Rhabdoid Tumor (AT/RT) Institutional Pathologist Pathologist Pathologist Diagnosis 1 2 3 MB (n = 42) 45 39 40 AT/RT (n = 26) 23 29 24 Institutional 3 of 3, At Least 2 of Diagnosis No.
Atypical teratoid/rhabdoid tumors (ATRTs) may form a histological spectrum from pure rhabdoid to atypical teratoid/ rhabdoid tumors, (87,88) and occur most commonly in young children (Figure 4).
There were 136 glioma (including 21 high-grade gliomas, 15 pilocytic astrocytomas, 19 medulloblastomas, 17 atypical teratoid/rhabdoid tumors, and 64 ependymomas) samples and 13 normal samples in our study.
Rorke, "Alterations of the hSNF5/INI1 gene in central nervous system atypical teratoid/rhabdoid tumors and renal and extrarenal rhabdoid tumors," Clinical Cancer Research, vol.
Of the 26 IRG recipients, seven focus significantly on pediatric cancers, including Ewing sarcoma, rhabdoid tumors, and atypical teratoid/rhabdoid tumors (RT).
The information is presented in chapters covering benign peripheral nerve sheath tumors (neurofibromas, schwannomas, and perineuriomas); malignant peripheral nerve sheath tumors; meningioma; hemangioblastoma of the central nervous system; paraganglioma and pheochromocytoma; atypical teratoid/rhabdoid tumors of the central nervous system; neuroblastoma and related tumors; medulloblastoma, primitive neuroectodermal tumors, and pineal tumors; and key mechanisms and pathways.
(2) The deletion or loss of gene function can also be demonstrated by loss of the expression of related protein products, such as INI-1 in epithelioid sarcomas, renal medullary carcinomas, rhabdoid tumors, and atypical teratoid/rhabdoid tumors; E-cadherin in lobular breast carcinomas and some signet ring cell gastric carcinomas; and mismatch repair proteins (MutL homolog 1 [MLH1], MutS protein homolog [MSH]2, MSH6, and postmeiotic segregation increased 2 [PMS2]) in microsatel lite instability tumors.
(111,112) It has been shown previously that this protein is lost in other aggressive tumors, such as renal and extrarenal rhabdoid tumors, atypical teratoid/rhabdoid tumors of the central nervous system, and epithelioid sarcomas.
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