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B2ARBeta 2-Adrenergic Receptor (also seen as ADRB2)
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38), (39) These autoantibodies may act as agonists, "causing or enhancing peripheral vasodi lation (mediated by B2AR and/or M3R activation) or inhibiting compensatory rise in pulse rate (M2R).
These preclinical studies reinforced the rationale for investigating A2A and B2AR agonists in the treatment of multiple myeloma and other B-cell malignancies and continue to highlight the power of the cHTS technology to interrogate combination activity across large panels of cancer cells to identify novel mechanisms.