BRMEBuckeye Rail Model Engineers (Ohio)
BRMEBusiness Research Methods Enterprise (UK)
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In a spiking experiment combination of standards with BRME (1: 1) increased the peak area at the same retention time, suggesting that the peak similarly seen for standards is included in this extract (Figure 1).
The results demonstrate that pretreated animal with BRME did not produce any significant antinociceptive effect in the first phase compared to control group.
The anti-inflammatory activity of BRME against carrageenan-induced paw oedema in rat revealed time and dose-dependent inhibition (Table 1).
In this study all the doses of BRME effectively suppressed the oedema produced by histamine and serotonin, so it maybe suggested that its anti-inflammatory activity is possibly backed by its antihistaminic activity.
The anti-inflammatory activity of BRME was also measured on rat paw using arachidonic acid as the inducer.
The BRME produced significant (P < 0.05, 0.01 and 0.001) reductions in oedema development.
In the TPA-induced ear oedema, the BRME led to an inhibition of ear weight and thickness in a dose-dependent manner.
Chemically-induced vascular permeability (such as seen with acetic acid) causes an immediate sustained reaction that is prolonged over 24 h and its inhibition suggests that the BRME may effectively suppress the exudative phase of acute inflammation [43].
The BRME reduces the dye leakage into the peritoneum in dose dependent manner.
In the present study, significant decrease in the WBC count caused by all doses of BRME and indomethacin suggests that these two agents inhibit the migration of WBCs to the site of inflammation.