CBDLColumbia Basin Development League (est. 1964; Washington)
CBDLChronic Bile Duct-Ligated
CBDLCommon Bile Duct Lithiasis
CBDLCommon Bile Duct Ligature
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References in periodicals archive ?
To evaluate the activation of pulmonary PI3K/Akt signaling in CBDL rats, protein levels of p-Akt (the active form of Akt) were measured by western blotting.
Expression of HIF-1[alpha] in the pulmonary tissue of CBDL and sham-operated rats
As shown in Figure 4, pulmonary HIF-1[alpha] levels were markedly decreased two weeks after CBDL and restored at 4 weeks.
NF-[kappa]B activation in the pulmonary tissue of CBDL and sham-operated rats
Four weeks after CBDL, NF-[kappa]B staining was markedly increased in rats, accompanied by increased VEGF-A signals.
In this study, we used CBDL rats to simulate human HPS, specifically focusing on the effects of quercetin on pulmonary microvascular angiogenesis.
The numbers of e-NOS stained cells in collecting and distal ducts were similar in the two groups with CBDL (Groups B and C) and were not larger than those observed in Group A.
ALT was analyzed every week and the mean level was higher in CBDL groups than in the sham group (Figure 3(i)).
NAC treatment of CBDL rats prevented liver and renal injury.
CBDL has been reported to decrease antioxidant defenses and liver concentrations of glutathione (GSH) and increase free radical formation [40].
There was a significant decrease in collagen staining after treatment with NAC once a day for 4 consecutive weeks after CBDL. The increase in the concentration of ALT after CBDL was less in rats that received NAC suggesting reduced hepatic inflammation.
This study shows that NAC ameliorates the deleterious effects of CBDL in liver and kidneys, a protection that appears to be related to potentiation of e-NOS.