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CCR7C-C Chemokine Receptor 7 (gene)
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[5] Human genes: CD2, CD2 molecule; CD3E, CD3e molecule, e (CD3-TCR complex); LCK, lymphocyte-specific protein tyrosine kinase; GZMA, granzyme A (granzyme 1, cytotoxic T-lymphocyte-associated serine esterase 3); CD28, CD28 molecule; MNDA, myeloid cell nuclear differentiation antigen; LYN, v-yes-1 Yamaguchi sarcoma viral related oncogene homolog; MYC, v-myc myelocytomatosis viral oncogene homolog (avian); BTG1, B-cell translocation gene 1, anti-proliferafive; CD79A, CD79a molecule, immunoglobulin-associated a; FAIM3, Fas apoptotic inhibitory molecule 3; CCR7, chemokine (C-C mofif) receptor 7; CD48, CD48 molecule; HLA-DRA, major histocompatibility complex, class II, DR a; FCER2, Fc fragment of IgE, low affinity II, receptor for (CD23); CD52, CD52 molecule.
We initially utilized the most common T cell memory definition based on CCR7 and CD45RO.
In the present study, we aimed to investigate whether BTLA overexpression was sufficient to preserve immune tolerance in imDCs with exogenous CCR7 overexpression.
Ectopic expression of Ascl2 upregulates CXCR5 but not bcl-6, and down regulates CCR7 expression in T cells in vitro, as well as accelerates T-cell migration to the follicles and Tfh cell development in vivo in mice.
Stimuli Increased secretomes Reference Hypoxia Tropomyosin, VEGF, SDF-1, [88, 90-93] FGF-2, HGF, IGF-1, Oct-4, Rex-1, and IDO IFN-[gamma] IDO, PGE-2, and Gal-9 [93-96] TNF-[alpha] Gal-9, BMP-2, VEGF, SDF- [91, 96, 97] 1, FGF-2, HGF, and IGF-1 TLR signal Gal-9 [96] Inflammatory stimuli CXCL-16, GRO, ENA-78, (IL-1[beta], IL-6, MIP-1-delta, [98] IL-20, and TNF- osteoprotegerin, MCP-1, [alpha]) MCP-2, MCP-3, IL-6, GCP- 2, and IL-2RA LPS VEGF, SDF-1, FGF-2, HGF, [91] and IGF-1 3D culture TSG-6, STC1, TRIL, IL-24, [100-102] and CD82 Nanosilicate More than 4000 gene [103] expression change Aging Increase: miR-335 [85, 86] Decrease: TNFR, IFNGR, CCR7, miR-146a, miR-155, and miR-132 TGF-[beta] Cytoskeletal factors (e.g.
BM-MSCs can migrate and adhere via CCR7, ICAM1-, VCAM1-, and Akt-dependent mechanism and enhance angiogenesis through increasing VEGF, NGF, BDNF, VEGF-A, eNOS, and HIF.
We demonstrated that activation of CCR7 by CCL21 can induce increased cell viability and mammosphere forming ability of colorectal cells via AKT/GSK-3[beta] signals.
Trogocytosis-mediated transfer of the chemokine receptor CCR7 was designed to occur from engineered K562 cells to the surface of human NK cells, resulting, in a study by Somanchi et al.
Gel electrophoresis revealed bands corresponding to the expected sizes of CCR1, CCR3, CCR4, CCR8, V28, and CCR11, while there were no bands for CCR2, CCR5, CCR6, or CCR7 (Figure 2).
In the rASC-based hydrogel treated group, muscle differentiation-related mRNAs, such as dystrophin, CCR7, myogenin, MyoD, and CTGF, were upregulated.
Walker et al., "Chemokine receptor 7 (CCR7) gene expression is regulated by NF-[kappa]B and activator protein 1 (AP1) in metastatic squamous cell carcinoma of head and neck (SCCHN)," The Journal of Biological Chemistry, vol.