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BJ cells treated with NT (10 nM) significantly increased the expression of EGF and IL-8 by 1.07 [+ or -] 0.45-(* P < 0.05, n = 3) and 0.88 [+ or -] 0.48-(* P < 0.01, n = 3) fold above control, respectively, while decreased CCL4 expression by -0.59 [+ or -] 0.27-(*P < 0.01, n = 3) fold below the control (Figure 4(a)).
Mouse CCL4, CXCL9, and CXCL10 are lyophilized, available in convenient sizes (5 and 25 micrograms) and in-stock ready to ship.
CCL4 (FC: -2.14) is also a chemokine protein, which is a chemoattractant for natural killer cells, monocytes, and a variety of other immune cells; it gets secreted and has chemokinetic and inflammatory functions.
Group II (n = 10) rats were injected with 40% CCl4 (the mixture of CCl4 and olive oil) in a standard normoxic chamber (Fi[O.sub.2] 0.21).
CCL4, INFa2, INF[gamma] and G-CSF had a higher release in low [Ca.sup.2+] condition, whereas, on the opposite, in high [Ca.sup.2+] concentration, a comparable or higher secretion of CCL2, CCL3, CCL5, CCL7, CCL11, CCL22, CXCL1, CXCL10, TGFa, TNF-[beta], IL-10, and IL-15 was seen.
The precipitate obtained were treated with N-bromosuccinamide (0.171 g, 0.98 mmol) in the presence of benzoyl peroxide as catalyst and carbon tetrachloride (CCl4) as solvent followed by washing with hot water to obtain N-(2-bromo-4-nitrophenyl)-3-hydroxy-4-oxo-3,4-dihydro-2H-benzo-[e][1,2]thiazine-3-carboxamide-1,1-dioxide (III).
against H2O2 and CCl4 induced hepatotoxicity in goat liver.
(36) to explain the observed reduction of liver fibrosis induced by CCl4 after the use of mesenchymal stem cells, because that study found no stem cells inside the liver.
Effects of kefir on (a) liver weights and (b) total body weights of mice treated with carbon tetrachloride (CCL4) after 4 weeks.
These cytokines include IL-1ra, IL-1[beta], IL-2, IL-2R[alpha], IL-3, IL-4, IL-5, IL-6, IL-7, IL-8, IL-9, IL-10, IL-12 (p40), IL-12 (p70), IL-13, IL-15, IL-16, IL-17, IL-18, CCL2, CCL3, CCL4, CCL5, CCL7, CCL11, CCL27, CXCL1, CXCL9, CXCL10, CXCL12, FGF, G-CSF, GM-CSF, HGF, IFN-[alpha]2, IFN-[gamma], LIF, [beta]-NGF, PDGF-BB, SCF, SCGF-[beta], TNF-[alpha], TRAIL, and VEGF.
More recently, several studies also suggest the potential antifibrotic effects of carvedilol in rats intoxicated with CCl4, those with ethanol-induced liver injury, and human umbilical vascular endothelial cell model.
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