CPT1Carnitine Palmitoyl Transferase 1
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The mRNA abundance of hepatic PEPCK, PC, CPT1, and PPARa, both prepartum and postpartum, was not affected by dietary energy density (p>0.05; Table 5).
Fenofibrate, another PPAR[alpha] agonist, is able to decrease body mass, independently of food intake [117], and to reduce visceral AT (VAT) mass [103] through PPAR[alpha] stimulation and upregulation of FA oxidation enzymes in AT, such as CPT1 [118, 119] and ACO [119].
In line #163, cytoplasmic lipids increased after treatment with etomoxir that blocks CPT1, transporter of long FAs into the mitochondrial membrane, thus affecting FAO.
The agent etoximir inhibitor of CPT1, responsible for mitochondrial import of fatty acids mediated by the carnitine shuttle, decreases intracellular ATP levels as well as cell viability in glioblastoma [109] and affects tumor growth in preclinical models [110].
Fenofibrate did not alter PPARa mRNA expression, but it reduced FAS mRNA expression and increased ACO, CPT1, and UCP2 mRNA expression in both HFD groups (Table 5).
Conclusion: AMPK activation not only elevated ATGL expression for lipolysis but also induced CPT1 expression for further mitochondrial translocation of fatty acids.
Acetyl-L-carnitine supplementation reverses the age-related decline in carnitine palmitoyltransferase 1 (CPT1) activity in interfibrillar mitochondria without changing the L-carnitine content in the rat heart.
This is a unique program that incorporates all 3 of the programs in which a successful graduate will receive a Medical Assistant National Certificate (MA), a State Certified Phlebotomy Technician Certificate (CPT1), and an Electrocardiography Certificate (EKG).
Victor Zammit and his team of researchers at the Warwick Medical School found that an enzyme known as "Carnitine palmitoyltransferase 1A" (CPT1) has a switch, which is thrown depending on the composition and curvature of its cellular membrane.
The CPT system incorporates two enzymatic complexes named CPT1 and CPT2.