The exact mechanism of combined CRAO
and CRVO has not been elucidated, but there are multiple mechanisms proposed to explain the association with retrobulbar injection.
[Treatment of Acute Central Retinal Artery Occlusion (CRAO
) by Hyperbaric Oxygenation Therapy (HBO)--Pilot study with 21 patients].
* Risk factors for CRAO
include cardiovascular disease, hypertension, diabetes, and other disorders associated with systemic inflammation.
A thorough medical examination was carried out to clarify the etiology of CRAO
. Carotid Doppler ultrasonography showed complete occlusion of the right common carotid artery (CCA) and internal carotid artery (ICA).
present a somewhat similar patient with combined CRAO
The incidence of stroke is said to be higher in patients with CRAO
. (2) Life expectancy is 5.5 years compared to people without CRAO
which is 15.
Numerous treatment modalities have been attempted in both CRAO
and BRAO without much success including low-intensity photocoagulation, intravenous prostaglandin El infusion, and enhanced external counter pulsation.
While visual loss due to central retinal artery occlusion (CRAO
) or branch retinal arterial occlusion (BRAO) following gas endotamponade and intraocular pressure (IOP) rise is a well-recognized complication [1,2], cases of central retinal venous occlusion (CRVO) or branch retinal venous occlusion (BRVO) after vitreoretinal surgery have not been reported so far.
The most likely etiology of a CRAO
in general and to this case is a cholesterol embolus (Hollenhorst plaque).
INTRODUCTION: Central retinal artery occlusion (CRAO
) is caused by closure of the central retinal artery by a thrombus or emboli.
Many systemic and ocular factors may cause acute central retinal artery occlusion (CRAO
Retinal arterial occlusions (RAO) may be divided anatomically into central (CRAO
) or branch (BRAO) forms, depending on the site of obstruction.