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The aim of the study was to investigate the ability of cTnT, CK and hsCRP in predicting the long-term risk of developing systolic dysfunction after 12 months in the population hospitalized with acute STEMI with previously normal left ventricular ejection fraction (LVEF) and with single-vessel coronary disease undergoing complete and successful revascularization.
The myocardial enzymes, cTnT and hepatic and renal function of patients in the three groups were detected and compared.
Although cTnT and cTnI belong to the same protein complex, they are structurally unrelated, which suggests that physical activity in general affects certain assays.
 Current studies employ cTnT, as it is universally standardised.
The cardiac Troponin T (cTnT) has been found to have excellent sensitivity and specificity and is superior to creatine kinase-MB (CK-MB) as indicator of myocardial necrosis.
The newest generation of cTnT assays has a 10- to 100-fold reduced limit of detection, below the 99th percentile of a normal reference population, and these assays are being adopted by a growing number of medical institutions.
cTnT (l) and CX43 (m) expression were detected on day 7 and significantly increased by day 14 (control = MSCs; null = [MSCs.sup.null] = MSCs infected with mock lentiviral vectors without miR-1; miR-1 = [MSCs.suip.miR-1] = MSCs infected with miR-1 recombinant lentiviral vectors; compared to MSCs, * P < 0.05, * P < 0.01; compared to [MSCs.sup.null], # P < 0.05, [??] P < 0.01; compared to [MSCs.suip.miR-1] (1 d), & P < 0.05, [??] P < 0.01; compared to [MSCs.suip.miR-1] (7d), @ P < 0.05).
Hemolysis is known to cause interference in the measurement of both cTnl and cTnT assays.
Assessing cTnT's role as a risk predictor became possible with the recent availability of high-sensitivity assays.
Although previous work showed an association between cTnT levels and heart disease, standard tests for the protein can detect cTnT in only a small percentage of the population, limiting the test's usefulness for assessing risk in people with no symptoms of heart disease.
People with detectable levels of cTnT were almost seven times more likely to die from heart disease within six years than those whose test results were negative.
The study team found that people with detectable levels of cTnT were almost seven times more likely to die from heart disease within six years.
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