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FW: 1958, HW: 1509, F: 379, T: 708, t1: 226, t2: 89, ctt1: 17, Mx4: 85, f1: 201, f2: 177, f3: 159, f4: 128, f5: 94, IO: 289, D: 201, d: 130, IO/d: 2.22, PO: 0.64.
Meantime, the mRNA expression of both CTT1 and CTA1 was significantly downregulated in MET18-deficient cells (Figure 2(c)).
Either CTT1 or CTA1 Restores the MET18 Deficiency-Suppressed Oxidative Stress Resistance in Yeast.
CTT1, but Not CTA1, Restores the Lifespan of MET18Deficient Yeast.
Our results suggest that MET18 deficiency increases the cellular response of yeast to [H.sub.2][O.sub.2] and CHP and shortens RLS through downregulation of catalases CTT1 and CTA1.
In our study, the total catalase activity as well as the mRNA levels of both CTT1 and CTA1 was significantly decreased in MET18/met18[DELTA] mutant compared with the WT strain (Figures 2(b) and 2(c)), resulting in an increased level of intracellular [H.sub.2][O.sub.2] in mutant cells (Figure 2(d)).
The DEX-R score correlated significantly with errors committed in both CTT1 (r = .345, p = .005) and CTT2 (r = .344, p = .005).
The five factor scores correlated significantly with errors in the CTT1 and CTT2.
Correlation between factor scores of DEX-R and neuropsychological tests Factor 1 Factor 2 Factor 3 Factor 4 CTT1 (N = 75) -0.157 -0.066 0.083 -0.117 n.s.
FW: 1477, HW: 1129, F: 362, T: 614, t1: 212, t2: 88, ctt1: 13, f1: 193, f2: 159, IO: 289, D: 170, d: 78, IO/d: 3.7, PO: 0.45.
FW: 1,149, HW: 200, Mx4: 216, F: 752, T: 1,126, t1: 498, t2: 167, ctt1: 2, IO: 448, D: 187, d: 134, IO/D: 2.39, PO: 0.71.
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