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References in periodicals archive ?
Cloning and characterization of two promoters for the human calcium-sensing receptor (CaSR) and changes of CaSR expression in parathyroid adenomas.
Amino-glycosides induce acute cell signaling and chronic cell death in renal cells that express the calcium-sensing receptor. J Am Soc Nephrol 2005; 16: 1236-1244, doi: 10.1681/ ASN.2004080631.
The extracellular calcium-sensing receptor (CaSR) belongs to class C of GPCR that signals in response to [Ca.sup.2+] and other ligands, such as gadolinium, polypeptides, and certain antibiotics [21, 22].
Familial hypocalciuric hypercalcemia with a de novo heterozygous mutation of calcium-sensing receptor. Endocrinol Diabetes Metab Case Rep 2015;2015:150016.
Recent data support the hypothesis that in vivo external calcium, through activation of calcium-sensing receptors, modulates the expression/trafficking of aquaporin 2 (AQP2).
Kim et al., "Significance of calcium-sensing receptor expression in gastric cancer," Scandinavian Journal of Gastroenterology, vol.
[5] SLC12A3, solute carrier family 12 (sodium/chloride transporter), member 3; CLCNKB, chloride channel, voltage-sensitive Kb; SLC12A2, solute carrier family 12 (sodium/potassium/chloride transporter), member 2; KCNJ1, potassium channel, inwardly rectifying subfamily J, member 1; CLCNKB, chloride channel, voltage-sensitive Kb; BSND, barttin CLCNK-type chloride channel accessory } subunit; CASR, calcium-sensing receptor.
[sup][18] identified Testin can also interact with calcium-sensing receptor (CaR), and influence cytoskeletal function via enhancing the CaR-mediated Rho signaling pathway.
A product with a different mechanism of action, cinacalcet, increases the sensitivity of the calcium-sensing receptor on the thyroid gland, which through a cascade of events prevents bone-related metabolism disorders.
Nevertheless, Vyleta and Smith (2011) reported that extracellular [Ca.sup.2+] does not trigger spontaneous glutamate release by simply increasing calcium influx but stimulates the calcium-sensing receptor and thereby promotes resting spontaneous glutamate release.
The medications evaluated were vitamin D analogs (paricalcitol and calcitriol), phosphate binders (calcium acetate and sevelamer), and a calcium-sensing receptor sensitizer (cinacalcet).
Natpara was not studied in patients with hypoparathyroidism caused by calcium-sensing receptor mutations or in patients with acute post-surgical hypoparathyroidism.
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