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The Co-IP assay revealed that the increase in PGC-1[alpha] acetylation caused by sirtinol (a SIRT1 inhibitor) considerably reduced the extent of the PGC-1[alpha]-PPAR[alpha] interaction in the kinsenoside-treated adipocytes (Fig.
electrophoretic mobility shift assay; Chip, chromatin immunoprecipitation; Co-IP, coimmunoprecipitation.
To examine the interaction between Rheb and FKBP38, we performed a co-IP experiment with anti-Rheb, observing that Rheb and FKBP38 were bound together in eluates from cotransfected cells (Figure 1B).