CB3

(redirected from Coxsackievirus B3)
AcronymDefinition
CB3Community Board 3 (New York City)
CB3Coxsackievirus B3 (infectious diseases)
CB3Cecropin B3 (biochemistry)
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References in periodicals archive ?
Enterovirus D68 was the most frequently reported type (56%); echovirus 30, coxsackievirus A6, echovirus 18, and coxsackievirus B3 were also frequently reported.
Many viruses have been implicated as causes of myocarditis, including coxsackieviruses group B, parvoviruses, echoviruses, adenovirus, influenza virus H1N1, Epstein-Barr virus, rubella (German measles) virus, varicella (chickenpox) virus, mumps virus, measles virus, yellow fever virus, dengue fever virus, polio virus, rabies virus, hepatitis A and C viruses, human immunodeficiency virus (HIV), and Zika virus; while parvovirus B19 (PVB19) has recently be demonstrated by endomyocardial biopsy (combined with polymerase chain reaction and in situ hybridization) as the most frequently detected virus in myocarditis, coxsackievirus B3 (CVB3) remains the most extensively studied virus that causes myocarditis both in human beings and in animal models [5-10].
Viral myocarditis (VMC) is the inflammation of cardiac muscle which is mostly due to viral infection such as coxsackievirus B3 [1].
Twelve EV-B serotypes were identified: echovirus 16 (E-16) and E-18 (5 each); E-11 and coxsackievirus B3 (CV-B3) (4 each); CV-B1, CV-B2, CV-B4, CV-A9, and E-6 (2 each); and E-3, E-5, and E-25 (1 each).
This earlier in-vitro study involved a panel of diverse and unrelated viruses, including coxsackievirus B3, poliovirus, vesicular stomatitis virus, and human cytomegalovirus (Proc Natl Acad Sci USA.
Proteasome inhibition reduces coxsackievirus B3 replication in murine cardiomyocytes.
MiR-342-5p suppresses coxsackievirus B3 biosynthesis by targeting the 2C-coding region.
When mice were fed a selenium-deficient diet and then inoculated with a nonvirulent strain of Coxsackievirus B3, the virus mutated to become a virulent strain.
Rapid genomic evolution of a non-virulent coxsackievirus B3 in selenium-deficient mice results in selection of identical virulent isolates.
Tracy, "Coxsackievirus B3 infection and type 1 diabetes development in NOD mice: Insulitis determines susceptibility of pancreatic islets to virus infection," Virology, vol.
Animals were infected intraperitoneally with 5 x [10.sup.5] plaque forming units (pfu) of Coxsackievirus B3 (CVB3) (Nancy strain) diluted in PBS.