DLX3Distal-Less Homeobox 3
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Let-7g-5p HMGA2 [69] miR-10b-5p Bcl6 [80] miR-19a Undetected [64] miR-19b Smad7 [43] miR-21 FasL, PDCD4 [41, 42] miR-22-3p HDAC6 [68] miR-23a Fas, Runx2 [70, 71] miR-24 Tcf-1 [81] miR-27 APC [56] miR-29a-3p DKK1, Kremen2, sFRP2 [82] miR-30 family Smad1, Runx2, LRP6 [59] miR-100 BMPR2 [83] miR-124 Dlx5, Dlx3, Dlx2 [73] NFATc1, RhoA, Rac1 [73] miR-125 ErbB2 [63] miR-140-5p BMP2 [65] miR-142 APC [60] miR-148a Kdm6b [46] MAFB [45] miR-2861 HDAC5 [75] Table 3: miRNA dysregulated in plasma of osteoporotic patients and their function.
Dlx2 and Dlx3 are expressed during tooth morphogenesis and have been shown to play a key role during cell differentiation and apoptosis.
Hue, "Expression and localization of DLX3, PPARG and SP1 in bovine trophoblast during binucleated cell differentiation," Placenta, vol.
(2011) reported that a SNP in DLX3 gene at 24 Mb of SSC12 was associated with drip loss in the same population as in this study, while a QTL for drip loss was detected at 28 Mb of SSC12 (Table 3).
Distinct roles for distal-less genes Dlx3 and Dlx5 in regulating ectodermal development in Xenopus.
Asada, "Association of DLX3 gene polymorphism and dental caries susceptibility in Japanese children," Archives of Oral Biolog, vol.
Roscigno et al., "A dominant mutation etiologic for human tricho-dento-osseous syndrome impairs the ability of DLX3 to downregulate ANp63a," Journal of Cellular Physiology, vol.