DIFL

(redirected from Direct Immunofluorescence)
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AcronymDefinition
DIFLDante Institute of Foreign Languages (India)
DIFLDual Induction Focused Log (petroleum drilling)
DIFLDubuque Independent Football League (Dubuque, IA)
DIFLDairy Investment Fund Limited (New Zealand)
DIFLDirect Immunofluorescence (microscopy)
DIFLDelhi International Football League (India)
References in periodicals archive ?
Direct immunofluorescence on cytological smears in oral pemphigus.
((c) and (d)) Direct immunofluorescence staining for IgA and C3 shows granular mesangial staining, respectively (400x).
Our patient's skin biopsy showed leukocytoclastic vasculitis with histopathologic evidence of IgA and IgM deposition based on direct immunofluorescence staining but renal involvement was absent.
Direct immunofluorescence (DIF) was consistent with a subepithelial bullous disease.
A case-control study was done using direct immunofluorescence on PAP smears in Hormozgan, Iran during 2004-2005.
In patients with active lesions, scrapings from lesions were collected and direct immunofluorescence was done using commercial kit of BioRad for confirming diagnosis of chickenpox.
The biopsy is generally made at the edge of the lesion, and direct immunofluorescence staining shows a linear deposition of IgG, IgA, or C3 along the basement membrane [8].
The diagnosis of disseminated discoid lupus erythematosus (DLE) was confirmed by histological and direct immunofluorescence examination.
Comparison of Chemicon SimulFluor direct fluorescent antibody staining with cell culture and shell vial direct immunoperoxidase staining for detection of herpes simplex virus and with cytospin direct immunofluorescence staining for detection of varicella-zoster virus.
Antinuclear antibody test, Direct Immunofluorescence study, and Herpes simplex virus types 1&2 antibody tests were negative.
Diagnosis of DH is established on the basis of a direct immunofluorescence test (DIF) revealing granular deposits of IgA in the papillae and the presence of circulating IgA antibodies directed against the endomysium and/or tissue and epidermal transglutaminase (tTG, eTG) [2, 3].
When detailed clinical data, such as location (face, arms, or chest), history of typical DLE, positive serologies, or direct immunofluorescence studies, are provided, the presence of only mild keratinocytic atypia in association with corroborative histologic findings (vacuolar change, follicular plugging, dermal mucin, superficial and deep infiltrate) enable the pathologist to make a definitive diagnosis of HLE.
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