This theory is supported by the fact that repression of EMT is
required for effective tumor initiation [24-27] and that CSC reprogramming often involves mesenchymal to epithelial transition (MET) [28, 29].
As a result, remodeling is now thought to be one of the main reasons for disease recalcitrance, and EMT is
receiving great attention as a convergence point between inflammation and pathological remodeling in many progressive fibrotic diseases.
The powertrain system configuration of the single-axle parallel PHEB with EMT is
shown in Figure 1.
Building on an earlier paper that showed FOXC2 is expressed more heavily in cells after EMT is
induced by a variety of factors, Mani and colleagues followed up first by using short hairpin RNA to suppress FOXC2 in breast cancer cells.
Ironically, it's the same process that enables certain aspects of embryonic development, but in the case of cancer it is far less beneficial "EMT is
characterized by loss of adhesion between cells and by increased migratory capacity," says Professor Howe.
"As far as we know, this is the first time such a test network has been opened in Eastern Europe and EMT is
also among the global pioneers in the field."
the most important step in this process," said Jin.
the leading operator in Estonia with 47 percent market share.
a morphologic cellular program simply defined as the phenotypic transition from an epithelial to a mesenchymal state.
Hypothesis: EMT is
important in cancer sternness and metastasis resulting in failure to differentiate; thus targeting EMT and related pathways can have clinical benefits.
increasingly recognized as an important pathway in the lung fibrosis, via giving rise to fibroblasts and myofibroblasts.
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