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For example, FAEEs were found to cause necrosis in pancreatic acinar cells by inducing sustained increases in free concentrations of [Ca.sup.2+] in the cytoplasm from released intracellular stores, leading to toxicity of mitochondria and failure to produce ATP (Criddle et al.
FAEEs by causing false negative results, fatty acid esters which may be
For the extraction of fatty acid methyl and ethyl esters (FAMEs and FAEEs), the official procedure reported in the Regulation EU number 61/2011 was applied to the whole set of samples.
FAEEs were synthesized using a short-chain fatty acid selective lipase.
FAEEs can be concentrated by use of hair products containing alcohol and both markers can be produced by a teetotal metabolism.
Also, low levels of FAEEs are found in the general population; thus a positive cut-off of 2 nmol/gram meconium is used with 100% sensitivity and 98.4% specificity.
Looking at the combined levels of four fatty acid ethyl esters (FAEEs) in hair provides an even longer-term measure of alcohol consumption.
Fatty acid ethyl esters (FAEEs) are formed in the body by esterification of ethanol with free fatty acids and can be detected in the serum and blood of alcohol users.
Derangements in fatty acid metabolism and transport and formation of fatty acid ethyl esters (FAEEs) also have been implicated in ethanol-induced cell injury.
The most widely studied putative biological biomarker is fatty acid ethyl esters (FAEEs) in the meconium (1) of newborns.
The esterification products of ethanol and fatty acids are known as fatty acid ethyl esters (FAEEs) (1-3).
Fatty acid ethyl esters (FAEEs) are nonoxidative metabolites that are formed when alcohol conjugates to endogenous free fatty acids and fatty acyl-CoA (Laposata 1998).
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