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FOSBFront-Opening Shipping Box (container for wafers)
FOSBFylde Office Service Bureau (UK)
FOSBFriends of St. Bartholomew (UK)
FOSBFriends of St. Botolph (UK)
FOSBFriends of Salmon Bay (Seattle, WA)
FOSBFriends of Sauble Beach (Canada)
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References in periodicals archive ?
Subsequent studies failed to demonstrate the presence of a putative fusion gene until 2014, when Walther et al, (19) using cytogenetics, fluorescence in situ hybridization, messenger RNA sequencing, and real-time polymerase chain reaction, demonstrated a balanced t(7;19)(q22;q13) translocation resulting in the fusion of the SERPINE1 and FOSB genes in several cases.
The immunohistochemical panel is quite distinct: PHE expresses CD31, ERG, FLI1, and FOSB, whereas epithelioid sarcoma is negative for these antibodies.
Other early responders include the stress transcription factors NF-KB, AP-1, FOSB, and Elk-3.
In addition, genes located at the 19q13 are the BAX and BCL3 apoptosis related genes [39], genes involved in cell cycle progression that codify for cyclins, such as CCNE1, and genes codifying for transcription factors, such as FOSB [40], among others [41].
Treatment of neuronal cells with etoposide, a Top II inhibitor which induces Top II-mediated DSBs [230], has been found to promote upregulation of a subset of nIEGs including Fos, FosB, and Npas4 [12].
Moratalla, "ERK phosphorylation and FosB expression are associated with L-DOPA-induced dyskinesia in hemiparkinsonian mice," Biological Psychiatry, vol.
For example, we previously identified activator protein 1 (AP1) family members such as FOSB and JUND in plasma-treated blood cancer cell lines to be crucial [54].
Wei et al., "The effect of electroacupuncture on extinction responding of heroin-seeking behavior and FosB expression in the nucleus accumbens core," Neuroscience Letters, vol.
miR-338-3p COL1A1; FOSB; ADAMTS1; MMP9 miR-139-5p FOS Table 4: Top 10 significantly enriched pathways in KEGG pathway analysis.
Gastrodia elata Blume alleviates L-DOPA-induced dyskinesia by normalizing FosB and ERK activation in a 6-OHDA-lesioned Parkinson's disease mouse model.
However, during the second part of the experiment, mice that were given high-caffeinated drinks mixed with alcohol not only behaved in the same manner as mice who had been given cocaine but they had increased levels of the protein FosB, which scientists say is responsible for long-term changes in the neurochemistry of the brain.