FACO

(redirected from Fatty Acyl-CoA)
AcronymDefinition
FACOFinal Assembly and Check Out
FACOFatty Acyl-CoA (Coenzyme A) Oxidase
FACOFellow, American Chiropractic Orthopedists
FACOFabrication, Assembly & Check Out
References in periodicals archive ?
Fatty acyl-CoA reductase (FAR) catalyzes the reduction of fatty acyl-CoA to fatty alcohol, and FAR1 and FAR2 encode the reductase enzyme, which is needed for the first step of the wax biosynthesis.
Fatty acyl-CoA reductase, encoded by IDG033 in wheat and Ms2 in Arabidopsis, is the key enzyme for the production of fatty alcohols in plastids [48].
The team found that medium-chain fatty alcohols could be produced in yeast by targeted expression of fatty acyl-CoA reductase, or TaFAR, in the peroxisome of Saccharomyces cerevisiae.
ETC complex I and complex II, as well as other mitochondrial enzymes such as [alpha]-ketoglutarate dehydrogenase, pyruvate phosphate dehydrogenase, fatty acyl-CoA dehydrogenase, and glycerol 3-phosphate dehydrogenase, can produce [O.sub.2.sup.*-]as byproduct, releasing it within the mitochondrial matrix.
Yield of ATP during oxidation of one molecule of saturated fatty acid with 16 carbons of fatty acyl-CoA. Metabolic Enzyme catalyzing N.
Inouye, "Enzymatic and genetic characterization of firefly luciferase and Drosophila CG6178 as a fatty acyl-Coa synthetase," Bioscience, Biotechnology and Biochemistry, vol.
[31] found that in hTNF-[alpha] transgenic mice, hepatic triglycerides were enhanced and accompanied by reduced hepatic mRNA levels or activities of CPT-II, mitochondrial HMG-CoA synthase, peroxisome proliferator activated receptor [alpha] (PPAR[alpha]), and fatty acyl-CoA oxidase (FAO).
Free CoASH released from fatty acyl-CoA intermediates by the methylketone cycle could be used by acyl-CoA oxidase and the thiolases, enabling fatty acid oxidation to continue [8].
We investigated whether the age-related impairment of cardiac fatty acid catabolism occurs, at least partially, through diminished levels of L-carnitine, which would adversely affect carnitine palmitoyltransferase 1 (CPT1), the rate-limiting enzyme for fatty acyl-CoA uptake into mitochondria for beta]-oxidation.
The latter is an important mechanism in the heart where increased respiration results in a fall in mitochondrial acetyl-CoA, in turn leading to a fall in cytosolic malonyl-CoA, relieving inhibition of carnitine palmitoyl transferase 1 (CPT I) and thereby increasing transport of fatty acyl-CoA into the mitochondria for oxidation [45].
Once inside the mitochondria, the fatty acyl-CoA is degraded through four separate reactions.
This decrease in total activity in response to the olive oil- and corn oil-enriched diets but not to the coconut oil-enriched diet could be due to the inhibitory effect of long-chain unsaturated fatty acyl-CoA on the synthesis and/or stability of acetyl-CoA mRNA, similar to that observed in the weaned rats [29].