GABAARGamma-Aminobutyric Acid Type A Receptor (neuroscience)
GABAARGamma-Aminobutyric Acid A Receptor Channel
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DAT/autorad (normal) 2008 D2R/autorad (normal) D1R/autorad (normal) CB1/autorad (normal) TH/immunohistochemistry (normal) DA-HVA/HPLC (normal) Abbreviations: autorad, autoradiography; CB1, cannabinoid receptor 1; ChAT, choline acetyltransferase; DA, dopamine; DAR, dopamine release; DOPAC, 3,4-dihydroxyphenylacetic acid; D2R, dopamine receptor; FDG, fluorodeoxyglucose; GABAaR, [gamma]-aminobutyric acid A receptor; GAD, glutamic acid decarboxylase; GP, globus pallidus; im, intramuscular; iv, intravenous; mAChR, muscarinic acetylcholine receptor; MRS, magnetic resonance spectroscopy; ND, not determined; IME, norepinephrine; sc, subcutaneous; SN, substantia nigra; SNpr, substantia nigra pars reticulata; TH, tyrosine hydroxylase.
The molecular mechanism might be associated with competitively binding of dihydromyricetin to BZ sites on GABAARs [61].
In order to study ligand gated ion channels like g-amino butyric acid type A receptors (GABAARs) and glycine receptors (GlyRs) modeling data were used to design mutagenesis experiments aimed at the characterization of glycosilation sites, found to be altered in disease states [10-12].