GAS1


Also found in: Medical.
AcronymDefinition
GAS1Growth Arrest-Specific 1 (antibody; also seen as GAS-1)
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References in periodicals archive ?
RNA-Seq analysis revealed significant upregulation of CALDN4 (FC: 2.02), UBD (FC: 3.59), IFGBP2 (FC: 2.02), GAS1 (FC: 2.32), ORM1 (FC: 2.02), and SMPD3 (FC: 2.20).
The genes GAS1 (FC: 2.33), CLDN4 (FC: 2.02), ORM1 (FC: 2.34), and UBD (FC: 2.34), which are recognized as some of most potent inflammatory markers, were found to be significantly upregulated in the livers of mycotoxin-treated groups.
(t, m, n) Asynchronous GAS-Harns Asynchronous GAS1. Initially, the group manager (GM) selects k (where kt > N - 1) random polynomials with degree t - 1, [f.sub.l](x) = [a.sub.l,0] + [a.sub.l,1]x + ...
McMahon, "The Hedgehog-binding proteins Gas1 and Cdo cooperate to positively regulate Shh signaling during mouse development," Genes and Development, vol.
***p= < 0.001; Abbreviations: AR-androgen receptor, VP=ventral prostates, DLP=dorsolateral prostate, DHT=dihydrotestosterone Clu-clusterin, ODC=ornithine decarboxylase, OAZ= ornithine decarboxylase antizyme, AdoMetDC=ade-nosylmethionine decarboxylase, SSAT=spermidine/spermine N1-actetyl-transferase, H3 = histone H3, Gas1 = growth arrest specific gene 1, GAPDH = glyceraldehydes 3-phosphate dehydrogenase, CLU =clusterin.
The results reveal a highly stereotypic pattern of response to IR in human diploid fibroblasts that reflects primarily synchronization behind the [G.sub.1] checkpoint but with prominent induction of additional markers of [G.sub.0] quiescence such as GAS1. Key words: cell cycle checkpoints, DNA damage, gene expression, human fibroblasts, ionizing radiation, microarray.
Similarly, on chromosome 13 is a growth-arrest-specific-1 (Gas1) locus that is very near that for a QTL affecting DA in M7, M9, and M10.
Expression of miR-34 is enhanced in DN and miR-34 induces MC proliferation by targeting GAS1 [69].
Finally, Rapamycin treatment upregulated the expression of growth arrest specific gene 1 (Gas1) that has antiapoptotic properties [48].
Moreover and in response to plasma-mediated oxidative stress, HaCaT keratinocytes contained significantly increased levels of several growth factor transcripts (e.g., CSF2, GAS1, FGF6, and IGFs).