GCSFRGranulocyte Colony-Stimulating Factor Receptor
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References in periodicals archive ?
Considering the overexpression of Gcsf-Chr19, it is plausible to hypothesize that this molecule would functionally interact with its receptor, Gcsfr. Therefore, both the Cxcl8b/Cxcr2 and Gcsf-Chr19/Gcsfr signaling pathways would allow neutrophils to leave the caudal hematopoietic tissue and enter the bloodstream [16].
(b) After damage, the expression of Gcsf-Chr19 would increase and interact with its receptor, Gcsfr. Likewise, mRNA levels of Cxcl8b and Cxcr2 would increase, and both proteins would interact.