GIPRGigabit Ip Router
GIPRGastric Inhibitory Polypeptide Receptor (microbiology)
GIPRGlucose-Dependent Insulinotropic Polypeptide Receptor (physiology)
GIPRGreat Indian Peninsular Railway
GIPRGeneration Interconnection Process Reform (California Renewable Energy Transmission Initiative)
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References in periodicals archive ?
[8] Human genes: QPCTL, glutaminyl-peptide cyclotransferase like; GIPR, gastric inhibitory polypeptide receptor.
The report provides comprehensive information on the Gastric Inhibitory Polypeptide Receptor (GIP-R or Glucose-Dependent Insulinotropic Polypeptide Receptor or GIPR), targeted therapeutics, complete with analysis by indications, stage of development, mechanism of action (MoA), route of administration (RoA) and molecule type.
GIPR advances the Operational Control Segment's ability to protect data and infrastructure, enhance the sustainability of the system, and meet future GPS operational requirements.
Recent study proved that GIP regulated directly on the metabolism in adipocytes through a naturally occurring variant of GIPR (E354Q) [18].
Robert Lutjens, head of biology at Addex Pharmaceuticals, said that GIPR and GLP1R are present together in pancreatic and nerve cells, and their roles in Type 2 diabetes make them important targets for the development of new drugs.
It has also become clear that [GIP.sub.1-42] is the main GIP species to have activity through the GIPR. Deacon and colleagues demonstrated that [GIP.sub.3-42] bound the receptor with much less affinity than did [GIP.sub.1-42] and had no effect on cAMP accumulation (30).
Addex Pharmaceuticals (Swiss: ADXN.SW), an allosteric modulation-based drug discovery and development biopharmaceutical company, announced today that its scientists have demonstrated that, in the presence of GLP-1, glucagon-like-peptide-1 receptor (GLP1R) can form a heterodimer receptor complex with gastric-inhibitory-peptide- receptor (GIPR).
(64), who found that fasting-glucose-increasing alleles of the MADD, gastric inhibitory polypeptide receptor (GIPR), GCK, FADS, DGKB, PROX1, TCF7L2, SLC30A8, and C2CD4B were associated with either abnormal insulin processing or secretion, whereas GCKR and IGF1 were associated with oral glucose tolerance test-based disposition indices and [beta]-cell function (64).
GIP and GLP-1 could induce biological effects through the GIP receptors (GIPR) and GLP-1 receptors (GLP-1R).
In order to evaluate GIPR activation, HIT-T15 cells were incubated in serum-free medium supplemented with 1% BSA and then exposed for 5 min to 100 nmol/L GIP [29-31].