GLUT

(redirected from Glucose transporter)
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AcronymDefinition
GLUTGlucose Transporter
GLUTOpenGL Utility Toolkit
GLUTGigantic Lookup Table
GLUTOpen Graphics Library Utility Toolkit
GLUTGamma Lookup Table
GLUTGraphics Library Utility Toolkit (open software)
GLUTGeorgia Land Use Trend (geospatial data; est. 1973)
References in periodicals archive ?
Expression of conventional and novel glucose transporters, GLUT1, -9, -10, and -12, in vascular smooth muscle cells.
Translocation of the glucose transporter GLUT4 in cardiac myocytes of the rat.
Liu et al., "Exercise improved lipid metabolism and insulin sensitivity in rats fed a high-fat diet by regulating glucose transporter 4 (GLUT4) and musclin expression," Brazilian Journal of Medical and Biological Research, vol.
ER: endoplasmic reticulum; eEF1A: eukaryotic elongation factor 1A; GcK: glucokinase; GLUT2: glucose transporter 2.
In diabetic patients, the expression of intestinal glucose transporters has been reported to be 3- to 4-fold higher than healthy controls [3], and therefore, higher amount of glucose will be absorbed by these patients in a shorter period of time, leading to increased postprandial glycemia.
Shirotani et al., "Identification of two enhancer elements in the gene encoding the type 1 glucose transporter from the mouse with are responsive to serum, growth factor, and oncogenes," The Journal of Biological Chemistry, vol.
Gao et al., "Glucose transporter 5 is undetectable in outer hair cells and does not contribute to cochlear amplification," Brain Research, vol.
In terms of glucose transporter (GLUT) defects, CSF glucose and blood glucose levels were normal (>50%), and the other metabolic investigations also gave normal results.
(50.) Cao H, Graves DJ, Anderson RA (2010) Cinnamon extract regulates glucose transporter and insulin-signaling gene expression in mouse adipocytes.
Cinnamon extract and polyphenols affect the expression of tristetraprolin, insulin receptor, and glucose transporter 4 in mouse 3T3-L1 adipocytes.
We also set out to understand the molecular mechanisms involving the activity of the insulin receptor (IR), the IR substrates 1 and 2 (IRS1/2), the phosphoinositol-3-kinase protein (PI3K), and protein kinase B (Akt/PKB), and the regulation of glucose transporter isoform 4 (GLUT4) in response to acute and chronic resistance exercise.
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