We again used a galactic density [rho] = 3 X [10.sup.6] [(1 + z).sup.3] counts per cubic Glyr
. The two curves simulated for this cosmology have an average galactic radius of R = 40,000(1 + z) light years and R = 13,000(1 + z) light years, respectively.
These antibodies are directed against two categories of antigens: (1) intracellular antigens (Hu, Ma2, CRMP5, amphiphysin, etc.) and (2) cell surface antigens (the VGKC complex, NMDAR, AMPARs, GABABRs, mGluR5 receptor, GlyRs
The Universe mean density is obtained by multiplying this figure with the average galactic count per cubic Glyr. Using dopplerian redshits the galactic count density is 4.6 x [10.sup.6] counts per cubic Glyr, leading to a mean Universe density of 1.84 x [10.sup.-30] g/[cm.sup.3].
Using the galactic density in the nearby Universe from figure 2 expressed per cubic Glyr LTD, and the volume of the sphere of radius 14 Gly LTD, the number of galaxies in the visible Universe is estimated at 175 billion.
In the scenario using LTDs with the sampling method, the galactic count per cubic Glyr grows according to a steep slope (figure 2), without accounting for the effect of the expansion which should add up to this growth.
Table 1: Estimation of the number of galaxies in the visible Universe (radius 14 Glyr) using LTD distances and Euclidean distances.
Taurine-Induced Actions Were Mediated via GlyRs and Extrasynaptic [GABA.sub.A] Receptors.
So, here in this study we tested taurine in the presence of picrotoxin to characterize the type GlyRs activated by taurineon SG neurons of Vc.
Following this further, we also used another selective [GABA.sub.A]R antagonist, bicuculline, which follows the same pattern as picrotoxin does, that is, blockade of homomeric GlyRs .
Presumably, this blockade of synaptic currents were via heteromeric GlyRs, and outward shift of holding current was induced via extrasynaptic GlyRs.
Taurine acted as an agonist on both extrasynaptic homomeric and synaptic hetromeric GlyRs on the SG neurons.
Taurine has been demonstrated for its ability in modulation of synaptic transmission by activating GlyRs and/or [GABA.sub.A]Rs.