HPBFHigh-Protein Barley Flour
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Human primary bronchial fibroblasts (HPBFs) express PAR-1, -2, and -3 but not PAR-4, and gingipains-induced secretion of hepatocyte growth factor (HGF) is significantly inhibited by RNA interference targeted at PAR-1 and PAR-2 68, 69].
It has been found that thrombin and a PAR-1 APs stimulate proliferation of HPBFs [68] and that thrombin and FoxO factors functionally interact through PI3K/Akt-dependent FoxO phosphorylation leading to vascular SMC proliferation [89].
Agonists of PARs including proteases can modulate neutrophil transepithelial migration, provoke proliferation of HUVEC, HPBFs, vascular SMC, and keratinocyte, and induce murine wound healing, fibrin formation, and bronchial epithelial repair.