(redirected from Human Diploid Fibroblast)
HDFHierarchical Data Format
HDFHubble Deep Field
HDFHome Depot Foundation (est. 2002)
HDFHolland Dance Festival (est. 1987)
HDFHuman Development Foundation (Schaumburg, IL)
HDFHelp Development File
HDFHost Data Facility
HDFHdsl Dual Framer
HDFHand Dyed Fibers
HDFHeavy Duty Fabric (product feature)
HDFHigh Density Fibreboard
HDFHereditary Disease Foundation
HDFHastings Diversified Utilities Fund (also seen as HDUF; Australia)
HDFHard Drive File (computing)
HDFHuman Diploid Fibroblast (oncology)
HDFHalt Die Fresse (German)
HDFHigh-frequency Direction Finding
HDFHard Decision Feedback
HDFHighest Density First
HDFHungarian Defense Force
HDFHard Disk Failure
HDFHalogenated Dibenzofuran
HDFHigh Density Frame (Seicor)
HDFHalt Dich Fest (Germany)
HDFHikkaduwa Development Foundation (Sri Lanka)
References in periodicals archive ?
18) Studies have shown that oxidative DNA damage contributes to replicative cessation in human diploid fibroblast cells, but this process can be slowed with the use of antioxidants and spin-trapping agents such as alpha-phenyl-t-butyl nitrone (PBN), which can act like an antioxidant.
Oxidative DNA damage and senescence of human diploid fibroblast cells PNAS.
In this review, we summarize our recent studies on the effects of these mitochondrial metabolites and mitochondrial antioxidants (alpha-phenyl-N-t-butyl nitrone and N-tbutyl hydroxylamine) on the age-associated mitochondrial decay of the brain of old rats, neuronal cells, and human diploid fibroblast cells.
Tocotrienol-rich fraction prevents cell cycle arrest and elongates telomere length in senescent human diploid fibroblasts.
Human diploid fibroblasts cultured under normal conditions have been reported to develop an irreversible cell cycle arrest, referred to as replicative senescence (Serrano and Blasco 2001).
We have examined the effects of the naturally occurring dipeptide carnosine (beta-alanyl-L-histidine) on the growth, morphology, and life span of cultured human diploid fibroblasts.
However, arsenite disrupts normal mitotic progression and induces aneuploidy in human diploid fibroblasts (Yih et al.
The results reveal a highly stereotypic pattern of response to IR in human diploid fibroblasts that reflects primarily synchronization behind the [G.
Induction of DNA strand breaks by intermittent exposure to extremely-low-frequency electromagnetic fields in human diploid fibroblasts.
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