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ICAM-1Intercellular Cell Adhesion Molecule Type 1
ICAM-1Intracellular Adhesion Molecule One
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References in periodicals archive ?
Since chronic inflammation and microvascular changes may play pivotal roles for the pathogenesis and the progression of the disease, we hypothesized associations among adiponectin, TNF-[alpha], ICAM-1, and VCAM-1 in patients with AD.
This study was designed to investigate if type 2 diabetes affects ICAM-1 levels, which could impair the immune response against M.tb.
The inflammatory cells in plaques and their inflammatory products may be the cause for plaque instability and ruptures.[1] Studies found three novel inflammatory markers, including lipoprotein-associated phospholipase A2 (Lp-PLA2), intracellular adhesion molecule-1 (ICAM-1), and chitinase-3-like protein 1 (YKL-40), which were involved in the clinical prognosis and pathogenesis of CHD.[2],[3],[4],[5],[6],[7],[8],[9],[10] Nevertheless, the association between these three inflammatory markers and plaque stability and the severity degree of coronary artery stenosis need to be further studied.
Rabbit interleukins (IL-1, IL-2, IL-6, IL-7, IL8, and IL-18), ICAM-1, VCAM-1, CRP, TNF-[alpha], and P-selectin kits were purchased from Cusabio (China).
Increased ICAM-1 levels (MFI) on both subpopulations were found in patients compared with controls, and this upregulation was more pronounced on A Mo than on T Mo (P < 0.001).
(2) Adhesion molecules involved in leukocyteendothelial transduction: L-Selectin, E-Selectin, ICAM-1, PECAM, and VCAM-1
In all groups, 20 paraffin blocks belonging to the primary tumor in the breast were stained by ICAM-1, VCAM-1, Cyclin D1 and Cathepsin D.
The future of the assessment of ED is in determining the level of biomarkers, adhesion molecules (VCAM, ICAM-1, selectins), and VEGF, fibrinogen, thrombomodulin and CRP in the blood.
Ltd., Hubei, China, MCP-1; NEO Group Inc., MA, USA, ICAM-1; Cloud-Clone Corp., TX, USA) in accordance with the manufacturer's protocol.
We assessed the prognostic significance of the quantitative amount of MP and all blood vessels as well as vascular expression of CD133 and ICAM-1. The latter mentioned markers were preferred over classical endothelial markers, since blood vessels in GB may not contain endothelial cells.
CIHH Treatment Increased the Expression of VLA-4, VCAM-1, and ICAM-1 and Decreased the Expression of CD162 and CD164 in BMMSCs of AA Rats.
Protein Expression Analysis by Western Blot of gp91-phox, NF-kB, ICAM-1, vWF, CD31, eNOS, Caspase-3 (Cleaved Caspase-3), and c-Jun.