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However, this response include and interact with additional genes, such as Notch-1, NADPH-quinone oxidoreductase (NQO1), the aryl hydrocarbon receptor (AhR), the Jun dimerization protein 2 (JDP2), and perhaps epigenetic changes that may be involved in sensing stress and damage and that are known to participate in processes leading to cellular senescence.
Table 2/Measurement Model Results Measurement Items Standardized Weight Critical Ratio TMS1 0.900 (Fixed) TMS2 0.970 18.337 TMS3 0.871 14.757 SVV1 0.787 (Fixed) SVV2 0.958 11.086 SVV3 0.786 9-772 JDP1 0.859 (Fixed) JDP2 0.848 11.773 JDP3 0.853 11.867 RSE1 0.797 (Fixed) RSE2 0.810 10.187 RSE3 0.817 10.303 RSE4 0.809 10.176 RSE5 0.786 9.796 RSE6 0.692 8.338 FMP1 0.815 (Fixed) FMP2 0.778 9.698 FMP3 0.787 9.851 FMP4 0.751 9.262 FMPS 0.767 9.521 Fit statistics: chi-square = 270.829 (df = 160, p < 0.001), CMIN/DF = 1.693, CFI = 0.940, NNFI = 0.929, RMSEA = 0.074 Table 3/Hypothesized Paths Testing Path St.
Yokoyama, "Histone modification activities of JDP2 associated with retinoic acid-induced differentiation of F9 cells," Nucleic acids symposium series (2004), no.
Murata et al., "JDP2, a repressor of AP-1, recruits a histone deacetylase 3 complex to inhibit the retinoic acid-induced differentiation of F9 cells," Molecular and Cellular Biology, vol.
Yokoyama, "Transcriptional regulation of the c-jun gene by AP-1 repressor protein JDP2 during the differentiation of F9 cells," Nucleic Acids Research.
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