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References in periodicals archive ?
In our study, differential regulation of glutamate receptor ionotropic, kainate 3 (GRIK3) and voltage-dependent R-type calcium channel subunit alpha-IE (CACNA1E) in frontal cortex datasets could be therefore biologically relevant with the mentioned pathways in AD brain areas.
Decanis et al., "Neuronal activity-dependent increase of net matrix metalloproteinase activity is associated with MMP-9 neurotoxicity after kainate," European Journal of Neuroscience, vol.
Ander et al., "Brain and blood microRNA expression profiling of ischemic stroke, intracerebral hemorrhage, and kainate seizures," Journal of Cerebral Blood Flow and Metabolism, vol.
Overstimulation of glutamate receptors such as NMDA, AMPA, and kainate receptors can cause an influx of calcium ions into the postsynaptic membrane.
Our previous study showed that GluR5-containing kainate receptors (non-NMDA glutamate receptor) regulate the inhibitory synaptic transmission through endogenous glutamate; therefore, we tested how neonatal propofol and etomidate exposure affect endogenous glutamatergic tonic regulation to inhibitory synaptic transmission in P90 approximately rats.[sup][17] To determine whether NMDA receptors are involved in the tonic glutamatergic input on GABAergic neurotransmission, we tested the effect of AP5, a selective NMDA receptor antagonist, on sIPSCs.
showed that low-frequency stimulation in ANT could decrease the frequency of high-frequency oscillations and interictal spikes in hippocampus in kainate mouse model [55].
Ethanol inhibition of AMPA and kainate receptor-mediated depolarizations of hippocampal area CAT.
Patients were genotyped for the single nucleotide polymorphism (rs2832407) in GRIK1 (glutamate receptor, ionotropic, kainate 1), a large gene on chromosome 21, which "encodes the Gluk1 subunit of the kainate (glutamate) receptor" to evaluate its impact as a moderator of topiramate's effects on drinking and on the adverse effects of the drug.
Ionotropic glutamate receptors (NMDA or N-methyl-D-aspartic acid, AMPA or a-amino-3-hydroxyl-5-methyl-4-isoxazole-propionic acid, and kainate) form an ion channel pore that is activated when glutamate binds to the receptor (Traynelis et al., 2010).