L-NAMEG -nitro-L-Arginine-Methyl Ester
L-NAMEL-Nitro-Arginine Methyl Ester
L-NAMEN-Nitro-L-Arginine Methyl Ester
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L-NAME in combination with DECC did not change the negative inotropy, negative chronotropy and the decrease in coronary flow induced by DECC.
L-NAME potentiated the antidepressant action of the sub-effective dose of bilberry extract (250 mg/kg).
Using this economically important animal model, the objectives of present study were: i) to verify NO effect using SNP (NO donor) on steroid synthesis, growth and apoptosis in GC ii) to verify whether the effects of SNP are modulated by L-NAME (NOS inhibitor) and heamoglobin (NO scavenger).
The combined action of the inhibitors of NO and Ach have also been tested, and it was observed that the combined action of L-NAME and scopolamine, the muscarinic receptor antagonist resulted in an impairment of maze learning in rats (38).
The present study showed that CAL-induced vascular remodeling was greatly enhanced in both eNOS-KO mice and in WT mice treated with L-NAME compared with WT controls.
Similarly diastolic pressure increases from the administration of L-NAME were inhibited with carvacrol injections.
To test the specificity of the reaction, SOD, L-NMMA, and L-NAME were added to the zymosan/hemocyte suspension in some samples.
Pre-incubation with L-NAME (unspecific nitric oxide synthase inhibitor), indomethacin (unspecific COX inhibitor), ODQ (soluble guanylyl cyclase inhibitor), atropine (cholinergic receptor antagonist), TEA (unspecific potassium channel blocker) reduced relaxations induced by 1.
However, pretreatment of L-NAME (5 mg/kg, ip), nitric oxide synthase inhibitor caused a potentiation in the protective effect of Curcumin (P < 0.
intact rings preincubated for 30 min with NO synthase inhibitor L-NAME (100 [mu]mol/1), precontracted with NA, and cumulatively exposed to 4 doses of the extract; and