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To assess NO involvement in the pathophysiology of T1DM, we treated Biobreeding rats, an animal model of autoimmune diabetes, with the general NOS inhibitor LNMMA at early stages of the insulitis process.
In cases that developed resistant cardiogenic shock, despite intraaortic balloon pumping and percutaneous coronary intervention after myocardial infarction; the administration of LNMMA significantly decreased the mortality rate (8).
Production of NO is inhibited by the analog of L-arginine, [N.sub.G] monomethyl-L-arginine (LNMMA) (Hibbs et al.