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The LVET interval is calculated as the distance between B and X point of the ICG signal.
By linear regression analysis, we found that E/A, E/e'm, and LVET predicted 35.6% of the variability of the sST2 levels [(F (3.82) = 3.940, p = 0.011)].
SV = rho * [(L/ [Z.sub.0]).sup.2] * LVET * dZ/[dt.sub.max] where L is the distance (in cm) between the recording electrodes measured at the front, [Z.sub.0] is the baseline impedance (in ohm) between the recording electrodes, LVET is left ventricular ejection time (in msec), and dZ/[dt.sub.max] is the peak value (in ohm [sec.sup.-1]) of the impedance waveform, and Rho represents the resistivity of blood (a value of 135 ohm cm was used).
Left ventricular ejection time (LVET) is the interval (in msec) between the occurrence of the B wave and the X point of the dZ/dt signal.
No significant differences were found for blood pressure, LVET, and PEP reactions.
Left ventricular ejection time (LVET) was measured from the onset to the end of left ventricular (LV) outflow curve.
The IVRTL, IVCTL, and LVET values increased for both cigarette types.
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We hypothesized that a volume overload lesion prolonging LV ejection time (LVET) may reduce IMP despite LV dysfunction (LVD).
Results: When compared to normal subjects (0.357[+ or -]0.122), IMP was increased with LVD (0.604[+ or -]0.278 p<0.001) but was similar in AR+Normal EF patients due to isovolumic relaxation time (IRT) and LVET prolongation.
Conclusions: The IMP in AR+Normal EF patients was similar to normals due to IRT and LVET prolongation.
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