LVPWTLeft Ventricular Posterior Wall Thickness (cardiology)
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As shown in [Table 3], the changes in LVEDd, IVST, LVPWT, LVM, LVMI, LAd, LVESd, EDV, ESV, SV, EF, and FS, at baseline and 12 months after treatment, were similar among the three groups (all P > 0.05).{Table 3}
The patients in the three groups did not have significant differences in LVEDd, IVST, LVPWT, LVM, LVMI, LA, LVESd, EDV, ESV, SV, EF, and FS levels between baseline and 12 months after treatment (all P > 0.05).
where LVID indicates LV internal diameter, LVPWT the LV posterior wall thickness, and IVST the interventricular septal thickness [13, 14].
(1998): LVM = 1.04 [[(LVEDd + IVSDd + LVPWd).sup.3]-[(LVEDd).sup.3]], where LVEDd is left ventricular end-diastolic internal diameter, IVSDd is intraventricular septum thickness during diastole, and LVPWT is left ventricular posterior wall thickness.
Analysis of variance revealed a significant effect of endurance training on LVM p < 0.001(F = 18.3), LVMI p < 0.001 (F = 17.1), IVSDd p < 0.001 (F = 17.3) and LVPWT p < 0.01 (F = 8.7).
There was a significant correlation between oxLDL concentration and LVPWT (z-score) (R = 0.258; p = 0.038).
Noninvasive transthoracic echocardiogram was performed by a sonographer, and the following cardiac measurements were obtained: interventricular septum thickness (IVST), left ventricular end diastolic diameter (LVED), and left ventricular posterior wall thickness (LVPWT).
Compared with subjects with low risk for OSA, subjects with high risk had higher levels of 0.5 h- and 2 h-BG ( P < 0.001 and P < 0.05), higher SBP ( P < 0.001) and DBP ( P < 0.001), and higher IVST, LVED, and LVPWT ( P < 0.001).