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LUXLarge Underground Xenon Detector (dark matter detection)
LUXLeft Upper Extremity (anatomy)
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The clinical history of poor glycemic control, dysmetria and coordination loss within the left upper extremity together with the characteristic imaging findings of contralateral basal ganglia hyperdensity on CT and hyperintensity on T1-weighted imaging are characteristic for the diagnosis of nonketotic hyperglycemia-induced hemichorea-hemiballism (NKHH).
In the operating room, the entire left upper extremity was prepped, and an initial incision was made over the dorsum of the hand.
baumanii has been constantly isolated from the affected eye, right tibia wound (before and after amputation), and his left upper extremity wound.
Additional imaging of the left upper extremity confirmed a proximal radioulnar synostosis (Figure 4).
Generalized morphea is induration of skin starting as four or more individual plaques larger than 3 cm, that become confluent and involve at least two of the seven anatomical sites (head, neck, right upper extremity, left upper extremity, right lower extremity, left lower extremity, anterior trunk and posterior trunk).3,4 Within this group there are distinct clinical presentations which are: disseminated plaques morphea, pansclerotic morphea and eosinophilic fasciitis.
However, mere elevation of the left upper extremity promptly resulted in reocclusion of the vessel, due in part to the rather extensive thrombosis.
On admission, the patient was found to have a limited range of motion in his left upper extremity with rigidity and apparent contracture with swelling secondary to a deep venous thrombosis.
He had 3/5 strength in the left upper extremity and 4/5 on the right upper extremity.
A 51-year-old female patient was admitted to our clinic with a three-year history of transient and recurrent pain and swelling in her left upper extremity. She had been diagnosed with left conjunctival malignant melanoma with multiple metastases.
After the seizure, patient was found to have on examination, left facial droop, left upper extremity power of 3/5, left lower extremity power of 3/5, sensory neglect, dysarthria, perservation, and confusion.
His paralysis, which began in his left lower extremity, ultimately included his left upper extremity and the flexors of his right lower extremities.
Postoperatively we noted the left upper extremity was colder than the right one and at the 12th postoperative day the patient developed paresthesia and then increasing pain starting with the 15th postoperative day and finally muscular rigidity and functional impairment.